你关闭了浪费，还不如把分给我，没好心人帮你答复，就连进来都不给你留个字，只有我，呵呵。我的答案满意吗。关闭浪费还不如给我，呵呵。
不高。根据查询相关公开信息显示，underreview接受概率是百分之30，UnderReview的状态持续一两个月算正常。杂志（Magazine），有固定刊名，以期、卷、号或年、月为序，定期或不定期连续出版的印刷读物，根据一定的编辑方针，将众多作者的作品汇集成册出版，定期出版的，又称期刊，杂志的形成来源于罢工、罢课或战争中的宣传小册子。
SECONDHAND SMOKE[19] The studies didn't just point to the ill effects of smoking on those who smoke--non-smokers, too, are apparently affected by the smoke from their friends, family members and strangers who light up in their presence.[20] A steady stream of reports documented the statistical risks of contracting cancer or suffering from heart disease, even if you've never put a cigarette to your lips.[21] The American Heart Association last fall released a seven-year study showing that never-smoking spouses of smokers have more than a 20 percent greater chance of death from coronary heart disease than those who have never smoked who live with non-smokers. That study gave more impetus to the drive to make workplaces and other public areas smoke-free.[22] The effects of smoking are hard on the children of smokers as well, the studies say. Dr. Claude Hanet of the St. Luc University Hospital in Brussels, Belgium, said earlier this year that a baby born to a smoking mother "should be considered an ex-smoker",[23] Hanet's study cautioned that cigarette smoke was more detrimental with decreasing age.[24] And a University of Birmingham, England, study, published in the British Journal of Cancer showed a possible link between fathers who smoked and an increased incidence of cancels in their children, while studies in the U.S. showed a possible link between smoking and DNA damage.被动吸烟[19)研究不光指出了吸烟给烟民自己造成的危害，还指出了非吸烟者显然也会受到他们吸烟的朋友、家人或在他们面前吸烟的陌生人的伤害。[20]持续不断的报告通过统计数字证明，即使你从来就没有吸过烟，也有患癌症或心脏病的危险。[21]去年秋季，美国心脏协会发表丁一份历时7年的研究报告，该报告指出，烟民的配偶虽然从不吸烟，但是和那些与非吸烟者生活的配偶相比，他们死于冠心病的危险要高出20％。这项调查进一步推动了在工作场所和公共场所禁烟活动的开展。[22]调查还表明，烟民的孩子也深受吸烟之害。今年年初，比利时布鲁塞尔圣·卢卡大学医院的克劳德·哈内特博士说，吸烟的母亲生下的孩于“应被看作是有吸烟史的人”。[23]哈内特的研究提请人们注意：年龄越小，吸烟的危害越大。[24]《英国癌症杂志》发表的一份英格兰伯明翰大学的报告显示，吸烟的父亲与其孩子患癌症比率的增加可能有联系；同时美国的研究则显示吸烟和脱氧核糖核酸受损可能有关系。
You Know Smoking Kills Why Can't You Stop! How To Kick It By Tania A Prince Illness And Smoking It is estimated that approximately 50 million Americans smoke. 400,000 people are thought to die in America each year from smoking related causes. Yet even when smokers are presented with these figures they still smoke. In the UK, it is estimated that 1/4 of the populace smoke, approximately 13 million adults. As smoking decreases in the older population the trend is increasing amongst the young. Comparison of The Different Techniques For Stopping Smoking There are many techniques available to help you stop smoking. But which of these is the most effective. In a scientific comparison of differing techniques used for stopping smoking (conducted by researchers at the university of Iowa, America), New Scientist reported that: "Hypnosis is the most effective way of giving up smoking" It is interesting to note that companies that produce chemical products, such as nicotine patches, also offer psychological help packages. Physical And Psychological Addiction When a person develops an addiction there are two components to it. One of these is a physical addiction to the substances themselves. The other is a psychological dependence. Surprisingly enough breaking the physical addiction with cigarettes is very easy. In fact if you think about it you sleep through the night for about eight hours without thinking or waking up for a cigarette. Yet when you are awake you may be a chain smoker, lighting one after the other. If the physical need was that great, you would wake up in the night. Any physical need dissipates within days. Psychological need can go on for years. Hypnotherapists can eliminate with the psychological need. Which Is The Most Effective Form Of Therapy? There are many forms of therapy available today that all claim to help you give up smoking. The difference between them is the speed and ease with which they help. Another major difference is if they require will power in order to be effective. The most effective techniques do not require will power. Will power implies conscious effort. Habits and addictions are controlled by the subconscious mind. It is very difficult if not impossible to over power the unconscious mind by will power. A hypnotherapist works with the unconscious mind gaining its support. That is why smokers can generally stop incredibly quickly and easily when hypnosis is used. Acupressure or acupuncture is also a technique widely used to help people stop smoking. It can be very effective. EFT, (Emotional Freedom Technique) is a very advanced yet simplistic form of acupressure. Used with hypnosis or by itself it can be extremely effective and fast in helping a person quit. EFT can be used to address the psychological and physical parts of the addiction. It is such a simple technique that the client can be taught how to use it during the session and if they need to use it after, they have a back up tool to help them get the result they want, and stop smoking. I worked with a client a couple of years back to help her eliminate a chocolate addiction. It worked really well. When I phoned her up for feedback some time later she told me an interesting story. Her husband had recently given up smoking using the will power method. He was driving her "nuts". So finally getting fed up, she said, "come on" and taught him how to do the technique. At first he felt a little uncomfortable, because if you've ever used EFT it is so simplistic you have to think "how on earth does this work", well after he got over that thought he said to his wife, "your not going to believe this but it's working". She told me that was the moment when it really sunk in for her how powerful the technique was. Will Power Versus Smoking No contest, smoking will win every time. Some people actually think they have used will power when what they have really done is called reframing. Reframing is when you generate a shift in thinking about something. Reframing is the basis of all therapy. The thing is you generally can not just tell yourself to think smoking in a different way. It's as if something has to "click" and suddenly you feel an automatic shift in your thinking. A therapist who knows how to reframe, needs to get their timing right, but when you do the results can be spectacular. I generally do reframing whilst I'm using EFT it seems to work more effectively. A client I worked with recently told me how he had made so many half hearted attempts to stop smoking, because he really didn't want to stop. He was only doing it for the other members of his family. He liked smoking. After going to the doctor about a medical problem something clicked inside of him and he finally decided he wanted to stop for himself, for his health. (Note, he wanted too, but he still loved smoking) Even though he wanted to stop, he felt envious of others who were able to smoke. He tried to reason out that other smokers also probably wanted to stop, but it didn't make him feel less envious, because it hadn't clicked, he still needed to make an effort to think this and if you'd really been able to listen to his thoughts you'd have probably heard a big ....but....(I like smoking...you don't really want to stop, you just don't want to get ill), at the end of that thought. Working through the problem, it also became evident that he felt great loss at the thought of never being able to smoke again. He felt a tight feeling in his chest. The question becomes what was he losing? The nice feeling he had when he smoked. The one that relieved the tight feeling in his chest. The tight feeling that wouldn't be there if he hadn't smoked. Long Term Effectiveness Of Hypnosis T Von Dedenroth found that 94% of 1000 subjects were still not smoking 18 months after completion of hypnotherapy. 1000 Subjects were used in the trial. (American Journal of Clinical Hypnotherapy (1968)). New Scientist, Magazine (October 1992) A Personal Account, Stopping Smoking Using Hypnosis And EFT ".....(It's) normally such a battle with yourself (to give up smoking without hypnosis, but the) biggest thing I found was that I didn't think about them anymore. I had no cravings. I felt more confident and calm in situations. Before I had often felt anxious going out, but after the session I felt more level headed. Usually certain triggers set you off, (but) there was just nothing. Interesting thing was that the underlying reasons were dealt with, not just the smoking......It's just lovely ...even my health problems have sorted themselves out. My nails were always brittle and weak and always cracked- now they are a lot stronger and my lungs are better......generally overall I'm a lot better" JB , Macclesfield Why do people smoke? This is a very interesting question. There are many reasons why a person may begin to smoke, these include: Trauma Peer Pressure Rebellion Glamour When people are under extreme stress they often look for ways of alleviating the anxiety. This can often lead to them taking up smoking. One client I worked with only began smoking in her forties when her husband was struck down with a major illness. She was under massive ongoing stress. Stopping Smoking The Facts Within eight hours Oxygen levels in the blood return to normal and the concentration of nicotine is now halved Within 48 Hours Your sense of taste sharpens and nicotine is completely out of your body 10 Years After Stopping The risk of having a heart attack has now diminished to the same level as a non smoker[1]Before Dr. Luther L. Terry, then the Surgeon General of the United States, issued his office's first "Report on Smoking and Health" more than 30 years ago, thousands of articles had already been written on the effects of tobacco use on the human body. [2] Tobacco companies had countered the reports--which purported to show links between smoking and cancer and other serious diseases--with denials and competing studies. [3] So in 1964, Terry and his Advisory Committee on Smoking and Health knew they were stepping into a major pit of controversy when they announced "cigarette smoking is a health hazard of sufficient importance in the United States to warrant appropriate remedial action". [4] It was America's first widely publicized acknowledgment that smoking cigarettes is a cause of serious diseases. [5] But the issue wasn't settled in 1964, nor is it settled in 1997, despite literally thousands more studies--and litigation that has forced at least one tobacco company to admit what some activists say they knew all along: cigarette smoke is hazardous to your health. [6] More than 30 years--and more than 20 Surgeon General reports--later, the issue appears headed for settlement in the courtroom rather than the laboratory. [1]30多年前，在当时美国的卫生局局长路德·L·特里博士发表他上任以来的第一份“有关吸烟与健康的报告”之前，已经有成千上万篇关于吸烟对人体健康影响的文章发表了。 [2]烟草公司对这些旨在表明吸烟和癌症以及其他疾病之间有关系的报告采取抵制的态度——予以否认，并提出分庭抗礼的研究报告。 [3]所以，在1964年，特里和他的吸烟与健康咨询委员会就明白了，在他们宣布“吸烟危害健康，在美国应引起人们足够的重视，有必要采取适当的补救措施”时，他们就踏人了一个争论的大坑中了。 [4]那是美国第一次广泛宣传，承认吸烟会导致严重的疾病。 [5]但是这个问题在1964年没有得到解决，在1997年还是没有解决，尽管确实又进行了数千项研究——且提起的诉讼已迫使至少一家烟草公司承认一些禁烟活动积极分子的说法，其实也是他们一直都明白的：吸烟对人体有危害。 [6]30多年以来，有20多位卫生局局长提出报告，此后，这个问题似乎是走向法庭解决，而不是在实验室解决。 So what are the risks? Here's what tobacco's critics say: CANCER [7] The Centers for Disease Control and Prevention says cigarette smoking is responsible for 151,322 cancer deaths annually in the United States. Most of those--116,920--are from lung cancer. The CDC says men who smoke are 22 times more likely to die from lung cancer than non-smokers. Women who smoke are 12 times more likely to die from the disease. [8] Statistical studies have long shown that people who don't smoke live longer than people who do and scientists have seen statistically the correlation between smoking and incidences of lung cancer since the 1950s. [9] But a study earlier this year by Gerd Pfeifer of the Beckman Research Institute pinpointed specific carcinogens in cigarette smoke that target parts of a gene already known to be prominent in some cancers. [10] Pfeifer wrote in Science that cigarette smoke causes changes in the gene p53, which protects against cancer when normal but promotes cancer growth when mutated . [11] Another study, published by the American Cancer Society, said that low-tar cigarettes offered no relief from the potential of cancer, and in fact were responsible for a type of cancer that reaches deeper into lung tissue. [12] Other cancers are also affected by cigarette smoke. An American Cancer Society researcher reported earlier this year that smoking increased men's risk of dying of prostate cancer, while other studies have linked tobacco use to increased risk of other cancers, including throat, breast and bowel cancer. 那么吸烟究竟有什么危害呢?还是听听烟草批评家们的说法： 癌症 [7]疾病防治中心说，吸烟是导致美国每年151322位癌症患者死亡的罪魁祸首。这些死亡者中的大多数——116990人是死于肺癌。疾病防治中心认为，男性烟民死于肺癌的可能性是非吸烟者的22倍。女性烟民死于肺癌的可能性是非吸烟者的12倍。 [8]统计数据研究早就表明，不吸烟的人比烟民长寿，而且自20世纪50年代以来，科学家们通过统计资料，已经发现了吸烟和肺癌发病率之间的相互关系。 [9]而今年早些时候，贝克曼研究所的格尔德·普法伊费尔所作的一项研究确切地指出了卷烟烟雾中固有的致癌物，这些致癌物袭击的目标是一种基因的某些部分，人们已经发现，这种基因在一些癌症中很突出。 [10]普法伊费尔在《科学》杂志上写道，吸烟使p53基因发生改变，这种基因在正常情况下起着防癌的作用，但在发生突变时，就会加快癌症的发展。 [11]美国癌症协会发表的另一份研究报告说，尼古丁含量低的卷烟并不能减少患癌症的可能性，它们实际上是引起一种进入到肺组织深层的癌症的罪魁祸首。 [12]而其他的癌症也受吸烟的影响。今年年初，美国癌症协会的一份研究报告表明，吸烟使男性死于前列腺癌的危险增加了，而其他的一些研究把吸烟同患上其他癌症（包括喉癌、乳腺癌和结肠癌）的危险的增加联系到了一起。 CARDIOVASCULAR DISEASES [13] Smoking also has been linked time and again to cardiovascular diseases. Among these, the biggest killer is heart disease: according to the CDC, smoking triples the risk of dying from heart disease among middle-aged men and women. [14] Studies also show an increased risk of death from stroke, aneurysms, high blood pressure, and other cardiovascular illnesses. RESPIRATORY DISEASES [15] Smoking is cited as a risk for dying of pneumonia, chronic bronchitis, or emphysema. The CDC says people who smoke increase their risk of death from bronchitis and emphysema by nearly 10 times. OTHER ILLNESSES [16] A report recently published in the American Journal of Epidemiology suggested that smoking increased the risk of developing non-insulin-dependent diabetes mellitus (NIDDM) by more than three times. [17] Studies have pointed to smoking as a risk in vision loss among older people, mental impairment later in life, Alzheimer's disease and other forms of dementia. EFFECT ON PREGNANCY [18] Pregnant women who smoke can pass nicotine and carbon monoxide to their baby through the placenta. Research indicates this can prevent the baby from getting the oxygen and nutrients it needs to grow--potentially leading to fetal injury, premature birth, or low birth weight. According to the American Lung Association, smoking during pregnancy accounts for an estimated 20 to 30 percent of low birthweight babies, up to 14 percent of premature deliveries, and about 10 percent of all infant deaths. 心血管疾病 [13]吸烟还被一再地和心血管疾病联系在一起，其中最大的杀手是心脏病。根据疾病防治中心的说法，吸烟使中年男女烟民死于心脏病的危险增至3倍。 [14]研究还表明，死于中风、动脉瘤、高血压和其他心血管疾病的危险也有所增加。 呼吸系统疾病 [15]吸烟被引证为导致死于肺炎、慢性气管炎或肺气肿的一种隐患。疾病防治中心说，吸烟的人死于支气管炎和肺气肿的危险比不吸烟的人高出将近10倍。 其他疾病 [16]美国《流行病学杂志》最近发表的一份报告表明，吸烟使人们患非胰岛素依赖型糖尿病的危险增加了3倍多。 [17]研究指出，吸烟有导致老年人中的失明、晚年大脑损伤、早老性痴呆病和其他类型的痴呆病的危险。 吸烟对妊娠的影响 [18]抽烟的怀孕女性会通过胎盘把尼古丁和一氧化碳传给胎儿。研究表明，这会妨碍胎儿获得发育所需要的氧气和营养物质，这可能对胎儿造成伤害，引起早产或胎儿体重不足。根据美国肺器官协会的说法，估计有20％至30％的体重不足的婴儿，多达14％的早产以及约10％的婴儿死亡都是由于女性在孕期吸烟引起的。吸烟的母亲还有可能通过喂母乳把尼古丁传给婴儿。 SECONDHAND SMOKE [19] The studies didn't just point to the ill effects of smoking on those who smoke--non-smokers, too, are apparently affected by the smoke from their friends, family members and strangers who light up in their presence. [20] A steady stream of reports documented the statistical risks of contracting cancer or suffering from heart disease, even if you've never put a cigarette to your lips. [21] The American Heart Association last fall released a seven-year study showing that never-smoking spouses of smokers have more than a 20 percent greater chance of death from coronary heart disease than those who have never smoked who live with non-smokers. That study gave more impetus to the drive to make workplaces and other public areas smoke-free. [22] The effects of smoking are hard on the children of smokers as well, the studies say. Dr. Claude Hanet of the St. Luc University Hospital in Brussels, Belgium, said earlier this year that a baby born to a smoking mother "should be considered an ex-smoker", [23] Hanet's study cautioned that cigarette smoke was more detrimental with decreasing age. [24] And a University of Birmingham, England, study, published in the British Journal of Cancer showed a possible link between fathers who smoked and an increased incidence of cancels in their children, while studies in the U.S. showed a possible link between smoking and DNA damage. 被动吸烟 [19)研究不光指出了吸烟给烟民自己造成的危害，还指出了非吸烟者显然也会受到他们吸烟的朋友、家人或在他们面前吸烟的陌生人的伤害。 [20]持续不断的报告通过统计数字证明，即使你从来就没有吸过烟，也有患癌症或心脏病的危险。 [21]去年秋季，美国心脏协会发表丁一份历时7年的研究报告，该报告指出，烟民的配偶虽然从不吸烟，但是和那些与非吸烟者生活的配偶相比，他们死于冠心病的危险要高出20％。这项调查进一步推动了在工作场所和公共场所禁烟活动的开展。 [22]调查还表明，烟民的孩子也深受吸烟之害。今年年初，比利时布鲁塞尔圣·卢卡大学医院的克劳德·哈内特博士说，吸烟的母亲生下的孩于“应被看作是有吸烟史的人”。 [23]哈内特的研究提请人们注意：年龄越小，吸烟的危害越大。 [24]《英国癌症杂志》发表的一份英格兰伯明翰大学的报告显示，吸烟的父亲与其孩子患癌症比率的增加可能有联系；同时美国的研究则显示吸烟和脱氧核糖核酸受损可能有关系。 SECONDHAND SMOKE ADDICTION [25] Of all the diseases associated with smoking, addiction is perhaps the one that receives the least attention. But President Clinton declared nicotine an addictive drug last August. In March, the Liggett Group, makers of Chesterfield and Lark brand cigarettes, admitted that cigarettes were addictive and cause cancer and agreed to pay about $750 million total to 22 states that had filed suit to force tobacco companies to pay for Medicaid for smoking-related illnesses. [26] Scott Harshbarger, the Massachusetts attorney general and president of the National Association of Attorneys General, told reporters that the Liggett deal "will produce information that indicates major tobacco companies were fully aware that the product they were selling is addictive, that the product they were selling had great impact on public health". [27] Other tobacco companies are clearly none too keen on the Liggett deal. For them, nicotine remains what they call a harmless flavor enhancement. 烟瘾 [25]在所有和吸烟有关的疾病中，也许人们最容易忽略的就是吸烟上瘾。然而，去年8月，克林顿总统宣布尼古丁为使人上瘾的麻醉药物。今年（指2000--编者注）3月，生产切斯特菲尔德和云雀牌香烟的利格特集团承认吸烟使人上瘾并会引发癌症，并且同意向22个州支付总额为7．5亿美元的赔偿金，这22个州提起了诉讼，要求烟草公司向治疗与吸烟有关的疾病的医疗补助制度进行赔偿。 [26]马萨诸塞州首席检察官、全美首席检察官协会会长斯哥特·哈什伯杰对记者说，对利格特集团的处理“将带来这样一个信息，那就是，大烟草公司充分意识到了他们出售的产品会使人上瘾，他们出售的产品对大众的健康影响极大”。 [27]很显然，其他烟草公司中没有一家对利格特集团的处理感兴趣。对他们来说，尼古丁仍然是他们所称的无害的提味剂。
不高。根据查询相关公开信息显示，underreview接受概率是百分之30，UnderReview的状态持续一两个月算正常。杂志（Magazine），有固定刊名，以期、卷、号或年、月为序，定期或不定期连续出版的印刷读物，根据一定的编辑方针，将众多作者的作品汇集成册出版，定期出版的，又称期刊，杂志的形成来源于罢工、罢课或战争中的宣传小册子。
（部份，英文）：1. Long, M.J., et al., A single-center, prospective and randomized controlled study: Can the prophylactic use of lamivudine prevent hepatitis B virus reactivation in hepatitis B s-antigen seropositive breast cancer patients during chemotherapy?Breast Cancer Research and Treatment, 2011. 127(3): p. 705-712.2. Gong, C., et al., Up-regulation of miR-21 Mediates Resistance to Trastuzumab Therapy for Breast Cancer.Journal of Biological Chemistry, 2011. 286(21): p. 19127-19137.3. Chen, J.Q., et al., CCL18 from Tumor-Associated Macrophages Promotes Breast Cancer Metastasis via PITPNM3.Cancer Cell, 2011. 19(4): p. 541-555.4. Wang, Y.H., et al., GeneSearch Breast Lymph Node Assay for the diagnosis of sentinel lymph nodes of breast cancer - CBCSG-001a: China validation study.Ejc Supplements, 2010. 8(3): p. 154-154.5. Gong, C., et al., Markers of Tumor-Initiating Cells Predict Chemoresistance in Breast Cancer.Plos One, 2010. 5(12).6. Wang, Y.S., et al., China Validation Study of GeneSearch (TM) Breast Lymph Node Assay for the Diagnosis of Sentinel Lymph Nodes of Breast Cancer - CBCSG-001a Interim Results.Cancer Research, 2009. 69(24): p. 544S-545S.7. Rao, N.Y., et al., Evaluating the performance of models for predicting the BRCA germline mutations in Han Chinese familial breast cancer patients.Breast Cancer Research and Treatment, 2009. 116(3): p. 563-570.8. Rao, N.Y., et al., Models for predicting BRCA1 and BRCA2 mutations in Han Chinese familial breast and/or ovarian cancer patients.Breast Cancer Research and Treatment, 2009. 113(3): p. 467-477.9. Cao, A.Y., et al., The prevalence of PALB2 germline mutations in BRCA1/BRCA2 negative Chinese women with early onset breast cancer or affected relatives.Breast Cancer Research and Treatment, 2009. 114(3): p. 457-462.10. Cao, A.Y., et al., Mutation analysis of BRIP1/BACH1 in BRCA1/BRCA2 negative Chinese women with early onset breast cancer or affected relatives.Breast Cancer Research and Treatment, 2009. 115(1): p. 51-55.11. Wang, Y.S., et al., China multicenter study of sentinel node biopsy substituting axillary node dissection: CBCSG-01 trial.Ejc Supplements, 2008. 6(7): p. 170-171.12. Li, W.F., et al., The prevalence of BRCA1 and BRCA2 germline mutations in high-risk breast cancer patients of Chinese Han nationality: two recurrent mutations were identified.Breast Cancer Research and Treatment, 2008. 110(1): p. 99-109.13. Yu, F., et al., Iet-7 regulates self renewal and tumorigenicity of breast cancer cells.Cell, 2007. 131(6): p. 1109-1123.14. Hu, X.Q., et al., Stable RNA interference of ErbB-2 gene synergistic with epirubicin suppresses breast cancer growth in vitro and in vivo.Biochemical and Biophysical Research Communications, 2006. 346(3): p. 778-785.15. Gong, C., et al., A double-blind randomized controlled trial of toremifen therapy for mastalgia.Archives of Surgery, 2006. 141(1): p. 43-47.16. Su, F.X., et al., Psychological stress induces chemoresistance in breast cancer by upregulating mdr1.Biochemical and Biophysical Research Communications, 2005. 329(3): p. 888-897.17. Su, F.X., et al., Glutathion S transferase pi indicates chemotherapy resistance in breast cancer.Journal of Surgical Research, 2003. 113(1): p. 102-108.18. Song, E.W., et al., Intrasplenic transplantation of syngenic hepatocytes modified by IFN-gamma gene ameliorates hepatic fibrosis in rats.Transplant International, 2002. 15(9-10): p. 472-478.19. Song, E.W., et al., Blocking CTL-based cytotoxic pathways reduces apoptosis of transplanted hepatocytes.Journal of Surgical Research, 2001. 99(1): p. 61-69.20. Song, E.W., et al., Adenovirus-mediated Bcl-2 gene transfer inhibits apoptosis and promotes survival of allogeneic transplanted hepatocytes.Surgery, 2001. 130(3): p. 502-511.21. Song, E., et al., Rare occurrence of metastatic colorectal cancers in livers with replicative hepatitis B infection.American Journal of Surgery, 2001. 181(6): p. 529-533.(部份，中文)：[1]杨桦,李顺荣,萧俏珍,苏逢锡,. 麦默通系统在乳腺占位性病变中的诊疗作用[J]. 中国全科医学,2009,(21).[2]姚燕丹,黄松音,袁广卿,于风燕,龚畅,贾卫娟,吴畏,宋尔卫,苏逢锡,. Matrigel对不同Her2表达的乳腺癌细胞原位成瘤、增殖、凋亡和转移的影响[J]. 中国病理生理杂志,2009,(12).[3]杨桦,李顺荣,苏逢锡,. 影响保乳手术成功的若干因素分析[J]. 广东医学,2009,(9).[4]李顺荣,苏丹晨,萧俏珍,金亮,吴建南,苏逢锡,. 麦默通旋切系统切除乳腺癌13例[J]. 广东医学,2009,(8).[5]郭巨江,苏逢锡,姚和瑞,陈积圣,. 替代激活的单核细胞促进人乳腺癌细胞SKBR3的生长侵袭[J]. 南方医科大学学报,2007,(4).[6]贾海霞,苏逢锡,郭巨江,宋尔卫,贾卫娟,龚畅,胡孝渠,秦丽,. 乳腺癌新辅助化疗的坐标法定位[J]. 中华普通外科杂志,2006,(3).[7]胡孝渠,宋尔卫,陈积圣,李海刚,于风燕,贾卫娟,龚畅,郭巨江,苏逢锡,. 原位小鼠乳腺癌肝转移模型[J]. 广东医学,2006,(4).[8]胡孝渠,秦利,苏逢锡,姚和瑞,陈积圣,龚畅,郭巨江,于风燕,贾海霞,. 载体介导HER-2RNA干扰治疗乳腺癌研究[J]. 南方医科大学学报,2006,(5).[9]苏逢锡,胡孝渠,. 乳腺癌根治性手术后即刻乳房再造临床应用与评价[J]. 中国实用外科杂志,2006,(4).[10]贾卫娟,苏逢锡,龚畅,胡孝渠,郭巨江,秦丽. 乳腺癌患者非哨兵淋巴结转移预测因素的研究[J]. 中华普通外科杂志,2005,(7).[11]沈镇宙,柳光宇,苏逢锡,何萍青,杨名添,施俊义,盛援,邹强,李亚芬. 多西紫杉醇加表柔比星治疗局部晚期乳腺癌的多中心Ⅱ期临床研究[J]. 中华肿瘤杂志,2005,(2).[12]曾弘,李海刚,曾韵洁,贾渭娟,苏逢锡,. 乳腺叶状囊肉瘤21例临床病理分析[J]. 中山大学学报(医学科学版),2003,(S1).[13]贾卫娟,苏逢锡,陈积圣. 乳腺癌哨兵淋巴结活检126例的临床研究[J]. 中华普通外科杂志,2003,(3).[14]苏逢锡,巴明臣,周晓东,吴仙容,李燕,陈积圣,李君. 活体染料注射法识别乳腺癌患者前哨淋巴结失败及假阴性原因分析[J]. 中华肿瘤杂志,2002,(3).[15]苏逢锡,贾卫娟,何嘉辉,曾韵洁,李海刚,陈积圣. 乳腺癌哨兵淋巴结检出相关临床和组织学因素[J]. 中华外科杂志,2002,(3).[16]巴明臣 ,苏逢锡 ,周晓东 ,吴仙容. 活体染料注射法识别乳腺癌哨兵淋巴结失败及假阴性的原因分析[J]. 新医学,2002,(12).[17]苏逢锡!510120广州,曾韵洁!510120广州,贾渭娟!510120广州,陈积圣!510120广州. 卵巢癌乳腺转移一例病例报告[J]. 中华肿瘤杂志,2001,(2).[18]苏逢锡!510120广州,陈积圣!510120广州,贾卫娟!510120广州硕士研究生,宋尔卫!510120广州博士研究生. BCL-2基因表达与乳腺癌抗凋亡及免疫耐受的关系[J]. 中华普通外科杂志,2001,(9).[19]苏逢锡,贾渭娟,陈积圣,宋尔卫. 乳腺癌细胞可溶性FasL分泌及其对淋巴细胞凋亡的诱导[J]. 肿瘤,2000,(4).[20]苏逢锡!510120广州,贾渭娟!510120广州,李海刚!510120广州,曾韵洁!510120广州,陈积圣!510120广州. 哨兵淋巴结活检对预测乳腺癌腋窝淋巴结转移的价值[J]. 中华外科杂志,2000,(10).
prevent防止,预防treat对待，治疗。根据你问的这些词在语境中应该是治疗的意思special特殊的machines机器electricity电，电力diseases疾病cancer癌症travel旅行themselves他们自己size大小，尺码taste品尝，口味muscles肌肉actually事实上
中成药杂志在官网可以随时在线查询文章。中成药杂志曾用刊名中成药研究，是由上海市卫生局主管，国家食品药品监督管理局、信息中心中成药信息站、上海中药行业协会主办的学术期刊，目前以月刊的形式出版发行。中成药杂志不仅是科技核心期刊，也是北大中文核心期刊和CSCD核心期刊。中成药杂志等级较高，投稿难度相对较大，可适用于高级职称的评级晋升。
一个月未收稿转投其他期刊，结果发现前者开始受理稿件怎么一个月未收稿转投其他期刊，结果发现前者开始受理稿件怎么办,本人菜鸟一枚，求助各位同仁这种情况怎么办？一个月前我投了某期刊A，一直在“最新投稿”状态，编辑部一直没有收稿。我明确在期刊官网上看到“自投稿之日起，每个环节（最新投稿→初审→外审或复审→发采用通知）最长不多于
DMM被屏蔽了下个小语加速器就能上了
javascript出错了，点那个设定方法啊，不会的话继续戳进去截图过来
ai少女的数字版在DL官网与DMM官网，都是需要梯子的。实体版在日本亚马逊官网，玩家们可以根据自己的需求选择。DL购买想要购买数字版的玩家请先进入dl的官网(需要梯子)，在右上角切换年龄分级选项至r18，弹窗确认以后在上方的搜索框内输入ai*少女就能打开游戏售卖界面了。放入购物车，并按照提示步骤付费即可成功购买。另外本作在dl的现阶段销量居于首位，所以大家也能在dl首页右边的排行版榜单进入游戏界面。DMM购买首先进入日本DMM的官网网站(也要梯子)，点击左边的dmmsoftware进入pc软件版块，输入ai*少女后进入游戏界面购买即可。如果搜索不到是因为还没登录，所以数字版首推dl版本。亚马逊购买想要入手实体版的小伙伴请走亚马逊，先进去日亚官网，然后在首页搜索框里输入ai少女查找相关游戏，接着它会跳出年龄确认框，点击左边的はい后找到游戏界面进去，点击右边的购买按照提示支付就行。更多关于ai少女在哪个平台，进入：查看更多内容
《免疫学杂志》由中国免疫学会和第三军医大学主办，1985年创刊，现为月刊，A4开本，内文92页，铜板纸、彩图随文印刷，国际标准连续出版物号：ISSN：1000—8861，国内统一刊号：CN51—1332/R。本刊坚持以交流学术信息、开展学术争鸣、繁荣学术园地为其办刊办刊宗旨；为本学科及相关学科读者充实理论、更新知识、积累经验服务为其目的。主要栏目有免疫学进展、专家论坛、基础免疫学、临床免疫学、免疫学技术与方法、短篇报道等。1989年以来先后被美国化学文摘(CA)、俄罗斯文摘（AJ）、波兰哥白尼索引(IC)、中国科技论文统计源期刊(中国科技核心期刊) 、“中国科学引文数据库源期刊”、“中国医学文摘·基础医学”、“中国生物医学光盘数据库”、“中国学术期刊(光盘版)”、“中国生物学文摘”、“中国学术期刊综合评价数据库”、“万文数据库”等国内外10多家重要数据库及检索刊物作为来源期刊收录。《中国核心期刊要目总览》1991年第1版、1996年第2版、2000年第3版和2004年第4版均将《免疫学杂志》列为基础医学类核心期刊。1990年获四川省首届科技期刊评比编辑加工奖；1995年获重庆市首届优秀期刊评选二等奖；1997年获重庆市优秀期刊一等奖；1997年获全军医学期刊质量评比二等奖；1997年获中国科协优秀科技期刊三等奖；1999年获重庆市高校优秀学报(期刊)二等奖；1999年获全军医学期刊质量评比优秀奖；2000年获首届《CAJ-CD规范》执行优秀奖；2004年获全军医学期刊质量评比优秀奖。2005年被重庆市新闻出版局、重庆市科学技术委员会、重庆市科技期刊编辑学会评为重庆市科技期刊优秀编辑部。本刊公开发行，全国各地邮局订阅，国外读者可向中国国际图书贸易总公司(北京399信箱，邮编：100044)订阅；国内代号：78-32，国外代号：BM4316；每本订价15元，全年90元。也可直接来信邮购。编辑部地址：重庆市沙坪坝区高滩岩第三军医大学，邮政编码：400038。 基础免疫学临床免疫学免疫学技术与方法综述与讲座短篇报道简讯 中国科协优秀期刊三等奖全军医学期刊评比优秀奖重庆优秀期刊一等奖 主管单位：第三军医大学主办单位：第三军医大学；中国免疫学会主编：吴玉章ISSN：1000-8861CN：51-1332/R地址：重庆市沙坪坝区高滩岩邮政编码：400038
1， 期刊名：The Anatomical Recorda. 简介：The Anatomical Record是美国解剖学会的官方期刊，刊登形态学和进化生物学方面的原创性工作。其亚洲副主编是浙江大学的李继承教授。他欢迎意诺的客户选择The Anatomical Record发表学术论文。b. 特点：该杂志对中国科技工作者比较友好。其最新一期20篇论文里有2篇来自中国大陆。意诺的客户在该杂志的成功发表率是75%。c. 影响因子：1.5 (2009)d. 网站：http://onlinelibrary.wiley.com/journal/10.1002/(ISSN)1932-8494e. 投稿方式 （网上投稿）: ， 期刊名：Leukemia Researcha. 简介： Leukemia Research是由 Elsevier 出版，刊登与白血病研究和治疗有关的论文（基础研究或临床研究都可以）。b. 特点：该杂志很器重中国学者的科研工作。最新一期21篇论文里，有4篇来自于中国大陆（20%的比例），其中有1篇是意诺的客户。c. 影响因子： 2.37 (2009)d. 网站： http://www.elsevier.com/wps/find/journaldescription.cws_home/583/authorinstructionse. 投稿方式（网上投稿）： ， 期刊名：Cancer Lettersa. 简介： Cancer Letters是由 Elsevier 出版，刊登与癌症有关的基础或临床研究。论文被录用的重要标准之一就是该论文能吸引跨学科的科研工作者。Cancer Letters的副主编有6位来自中国大陆。b. 特点：如果您从事癌症方面的工作，这个杂志是您最佳的选择。最新一期11篇论文里，有5篇来自于中国大陆（50%的比例）。意诺的客户2010里在该杂志累计发表6篇论文。c. 影响因子：3.74 (2009)d. 网站： http://www.elsevier.com/wps/find/journaldescription.cws_home/506050/authorinstructionse. 投稿方式（网上投稿）： ， 期刊名：The Journal of Biological Chemistrya. 简介： The Journal of Biological Chemistry是由 美国生物化学和分子生物学会主办，主要发表生物化学和分子生物学方面的研究。副主编里有一位中国学者。b. 特点：最新一期有3篇论文来自于中国大陆，其中一篇是意诺的客户。c. 影响因子：5.33 (2009)d. 网站： http://www.jbc.org/site/misc/itoa.xhtmle. 投稿方式（网上投稿）： ， 期刊名：Brazilian Journal of Medical and Biological Researcha. 简介： Brazilian Journal of Medical and Biological Research是由 巴西科学会主办，主要发表医学和生物学方面的研究。b. 特点：推荐这个杂志的主要原因，一是对中国学者比较友好。最新一期8篇文章有3篇来自中国大陆；二是因为巴西的官方语言不是英语，该杂志对英语的要求没有那么苛刻。意诺客户在此杂志上的成功发表率是95%。c. 影响因子：1.08 (2009)d. 网站： http://www.scielo.br/revistas/bjmbr/iinstruc.htme. 投稿方式（电子邮件）： ， 期刊名：PLoS ONE a. 简介： PLoS ONE是全球最大，最好，影响因子最高的开放杂志，接受任何自然科学有关的论文。b. 特点：因为是开放杂志，在上面发表的论文能被更多的学者读到，从而大大提高您论文被引用的机会。此外，它有20多位编委来自中国。过去12个月里，意诺帮客户往此杂志上发表了13份篇论文。c. 影响因子：4.35 (2009)d. 网站： http://www.plosone.org/static/checklist.actione. 投稿方式（网络投稿）： ， 期刊名：The Journal of Immunology a. 简介： The Journal Immunology是美国免疫学协会的旗舰杂志，接收免疫学方面的基础研究，临床应用，以及政策法规方面的论文。b. 特点：接收率高（41%的接受率）。从接收到发表的时间少于2个月。此外对中国学者比较友好，最新一期有5篇论文来自中国大陆，2篇是意诺的客户。c. 影响因子：5.64 (2009)d. 网站： http://www.jimmunol.org/site/misc/authorinstructions.xhtmle. 投稿方式（网络投稿）： ， 期刊名：The FASEB Journal a. 简介： The FASEB Journal 是美国实验生物学会的旗舰杂志。b. 特点：对中国学者友好，最新一期有2篇论文来自中国大陆。意诺的客户在过去12个月里也在此期刊上发表过4篇论文。c. 影响因子：6.40 (2009)d. 网站： http://www.fasebj.org/site/misc/ifora.xhtmle. 投稿方式（网络投稿）： ， 期刊名：Proceedings of The Royal Society B (Biological Sciences) a. 简介： Proceedings of The Royal Society B是英国皇家学会的生物学期刊，发表生物学各方面的论文。b. 特点：中国区的编委是北京师范大学的张教授。意诺客户帮客户在此期刊上发表过5篇论文c. 影响因子：3.52 (2009)d. 网站： http://rspb.royalsocietypublishing.org/site/misc/information.xhtmle. 投稿方式（网络投稿）：